By U. Sjöbring, J.D. Taylor, A. Schmidt, H. Herwald
Exacerbations of bronchial asthma and COPD: definitions, scientific manifestations and epidemiology / O'Byrne, P.M. -- Human rhinovirus types in bronchial asthma / Singh, A.M.; Busse, W.W. -- Allergen inhalation problem: a human version of bronchial asthma exacerbation / Gauvreau, G.M.; Evans, M.Y. -- mobile and animals types for rhinovirus an infection in bronchial asthma / Xatzipsalti, M.; Papadopoulos, N.G. -- Modeling responses to breathing condominium airborne dirt and dust mite publicity / Cates, E.C. ... [et al.] --, breathing syncytial virus-induced pulmonary ailment and exacerbation of allergic bronchial asthma / Lukacs, N.W. ... [et al.] -- Lipopolysaccharide problem of people as a version for persistent obstructive lung disorder exacerbations / Kharitonov, S.A.; Sjobring, U. -- A human rhinovirus version of persistent obstructive pulmonary sickness exacerbations / Contoli, M. ... [et al.] -- Animal versions of cigarette smoke-induced continual obstructive lung ailment / Churg, A.; Wright, J.L. -- Animal versions of continual obstructive pulmonary sickness exacerbations / Gaschler, G.J. ... [et al.]
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Exacerbations of bronchial asthma and COPD: definitions, scientific manifestations and epidemiology / O'Byrne, P. M. -- Human rhinovirus types in bronchial asthma / Singh, A. M. ; Busse, W. W. -- Allergen inhalation problem: a human version of bronchial asthma exacerbation / Gauvreau, G. M. ; Evans, M. Y. -- mobile and animals versions for rhinovirus an infection in bronchial asthma / Xatzipsalti, M.
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Additional info for Models of Exacerbations in Asthma and COPD
J Allergy Clin Immunol 1997;100:669–678. Robinson D, Hamid Q, Bentley A, Ying S, Kay AB, Durham SR: Activation of CD4ϩ T cells, increased TH2-type cytokine mRNA expression, and eosinophil recruitment in bronchoalveolar lavage after allergen inhalation challenge in patients with atopic asthma. J Allergy Clin Immunol 1993;92:313–324. Ali FR, Oldfield WL, Higashi N, Larche M, Kay AB: Late asthmatic reactions induced by inhalation of allergen-derived T cell peptides. Am J Respir Crit Care Med 2004;169:20–26.
Modeling is an essential part of the scientific endeavor. In medicine, illness is manifested by a collection of signs and symptoms. An observer collects them, appraises them and decides to which recognized pattern they best fit. This pattern recognition exercise, essential in deriving a diagnosis, contributes only modestly to the understanding of the origin of a disease, its evolution and its nature. Origin, evolution and nature are facets of a complex process, especially in the case of chronic diseases such as asthma.
Wood LJ, Sehmi R, Dorman S, Hamid Q, Tulic MK, Watson RM, Foley R, Wasi P, Denburg JA, Gauvreau G, O’Byrne PM: Allergen-induced increases in bone marrow T lymphocytes and interleukin-5 expression in subjects with asthma. Am J Respir Crit Care Med 2002;166:883–889. Machado L, Stalenheim G: Factors influencing the occurrence of late bronchial reactions after allergen challenge. Allergy 1990;45:268–274. Lai CK, Beasley R, Holgate ST: The effect of an increase in inhaled allergen dose after terfenadine on the occurrence and magnitude of the late asthmatic response.
Models of Exacerbations in Asthma and COPD by U. Sjöbring, J.D. Taylor, A. Schmidt, H. Herwald